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Dietary fiber and incidence of type 2 diabetes in eight European countries: the EPIC-InterAct Study and a meta-analysis of prospective studies.
Consistent consumption of diets rich in fibers, particular vegetable and cereal fibers, may cut down type 2 diabetes risk.
This study investigated the relationship between the ingestion of diets rich in dietary fiber and the incidence of type 2 diabetes. Researchers analyzed the dietary and clinical data of 26,817 diabetic and non-diabetic subjects recruited from the European Prospective Investigative into Cancer and Nutrition (EPIC)-InterAct Study.
Researchers discovered that high total, vegetable, and cereal fiber intakes decreased the odds of developing type 2 diabetes. On the other hand, increased consumption of fruit fiber was found to have little or no effect on type 2 diabetes risk. The results of this meta-analysis reveal that regular consumption of high fiber diets may confer significant protection against the development of type 2 diabetes.
Research Summary Information
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2015
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Kuijsten A, Aune D, Schulze MB, Norat T, van Woudenbergh GJ, Beulens JW, Sluijs I, Spijkerman AM, van der A DL, Palli D, Kühn T, Wendt A, Buijsse B, Boeing H, Pala V, Amiano P, Buckland G, Huerta JM, Tjøonneland A, Kyrø C, Redondo ML, Sacerdote C, Sánchez MJ, Fagherazzi G, Balkau B, Lajous M, Panico S, Franks PW, Rolandsson O, Nilsson P, Orho-Melander M, Overvad K, Huybrechts I, Slimani N, Tumino R, Barricarte A, Key TJ, Feskens EJ, Langenberg C, Sharp S, Foroughi NG, Riboli E, Wareham NJ.
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InterAct Consortium
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Yes, Free full text of study was found:
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Yes. Source of funding disclosure found
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The InterAct project was supported by the European Union (LSHM-CT-2006-037197) in the Framework Programme 6; DA was supported by the Liaison Committee between the Central Norway Regional Health Authority (RHA) and the Norwegian University of Science and Technology (NTNU); TN was supported by the World Cancer Research Fund (WCRF); JWJB and IS were supported by the Netherlands Agency grant IGE05012 and Incentive Grant from the Board of the UMC Utrecht; DLVDA and AMWS were supported by the Dutch Ministry of Public Health, Welfare and Sports (VWS), Netherlands Cancer Registry (NKR), LK Research Funds, Dutch Prevention Funds, Dutch ZON (Zorg Onderzoek Nederland), World Cancer Research Fund (WCRF) and Statistics Netherlands; DP was supported by Associazione Italiana per la Ricerca sul Cancro-AIRC-Italy; TK and AW were supported by the German Cancer Aid, German Cancer Research Center (DKFZ Heidelberg) and German Federal Ministry of Education and Research; PA was supported by the Regional Basque Country Government; GB was supported by The Spanish Ministry of Health (ISCIII RETICC RD06/0020/0091) and the Catalan Institute of Oncology; AT and CK were supported by the Danish Cancer Society; MLR was supported by the Government of the Principality of Asturias; SP was supported by Compagnia di San Paolo; PWF was supported by the Swedish Research Council, Novo Nordisk, Swedish Diabetes Association and Swedish Heart-Lung Foundation; OR was supported by the Västerboten County Council; TJK was supported by the Cancer Research UK; NJW, SS, CL and NGF are supported by the Medical Research Council Epidemiology Unit MC_UU_12015/1 and MC_UU_12015/5.
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No. Potential conflicts disclosure not found
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